Prophage-encoded virulence factor, Gp05, contributes to endo…

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  • DOI 10.1128/mbio.01685-25
  • PMID https://pubmed.ncbi.nlm.nih.gov/40919817
  • OpenAlex ID W4414064362
  • Type Article
  • Language EN
  • Published 08 Sep 2025
  • Volume 16 (10)
  • Pages e0168525
  • Source mBio
  • Publisher American Society for Microbiology
  • FWCI 1.46
  • Countries 1
  • Institutions 5
  • Added 09 Sep 2025
  • Updated 27 Sep 2026
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Prophage-encoded virulence factor, Gp05, contributes to endothelial cell dysfunction and immune evasion to promote persistent methicillin-resistant Staphylococcus aureus endovascular infections

Article 2025 EN Gold

ABSTRACT Methicillin-resistant Staphylococcus aureus (MRSA) is a leading cause of endovascular infections, where interactions with endothelial cells play a critical role in pathogenesis. Gp05, a prophage-encoded protein, has previously been implicated in promoting antibiotic persistence by modulating MRSA cellular physiology and evading neutrophil-mediated killing. In this study, we investigated t…

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1.46

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6 authors
#AuthorInstitutionsORCID
1 Yi Li University of California, Los … (US) The Lundquist Institute (US) +1 —
2 Adhar C. Manna Dartmouth College (US)
3 Sarah Ibrahim University of California, Los … (US) The Lundquist Institute (US) +1
4 Richard A. Proctor University of Wisconsin–Madiso… (US)
5 Ambrose L. Cheung Dartmouth College (US)
6 Yan Q. Xiong University of California, Los … (US) The Lundquist Institute (US) +1
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